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|Title: ||Triclosan – an update|
|Authors: ||Saleh, S.|
Haddadin, R. N. S.
Collier, Phillip J.
|Affiliation: ||University of Abertay Dundee. School of Contemporary Sciences|
Mechanism of action
|Issue Date: ||Feb-2011|
|Type: ||Journal Article|
|Rights: ||Published version (c)Wiley-Blackwell, available from http://dx.doi.org/10.1111/j.1472-765X.2010.02976.x. The definitive version is available at www3.interscience.wiley.com|
|Citation: ||Saleh, S., et al. 2011. Triclosan – an update. Letters in Applied Microbiology. 52(2): pp.87-95. Available from http://dx.doi.org/10.1111/j.1472-765X.2010.02976.x|
|Abstract: ||The discovery in 1998 that triclosan has a site-specific action in the bacterial cell as an inhibitor of NADH- or NADPH-dependent enoyl-acyl carrier protein reductase led to a lively debate in the scientific press. The thesis of this debate was that such a mode of action may allow triclosan to induce resistance and cross-resistance in bacterial cells. The debate last saw review in 2004, and this paper aims at updating our knowledge in this area, given recent research on the topic.|
|Appears in Collections:||Science Engineering & Technology Collection|
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